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The pathological lesion is sclerosis (fibrosis) within the glomerulus and hyalinisation of the feeding arterioles, but no increase in the number of cells (hence non-proliferative) As the glomerulonephritis progresses, the tubules of the kidney become infected, leading to atrophy and hyalinisation. — “Glomerulonephritis - Wikipedia, the free encyclopedia”, en.wikipedia.org